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Related Articles from SNS
OpenAI and Anthropic Sign Letter to Prevent AI-Developed Biological Weapons
The CEOs of several major artificial intelligence companies are urging members of Congress to adopt new laws that would make it harder for bad actors to develop biological weapons using their technology. Google DeepMind’s Demis Hassabis, OpenAI’s Sam Altman, Anthropic’s Dario Amodei, and Microsoft AI’s Mustafa Suleyman are among the signatories on a public letter calling for laws requiring companies that sell synthetic DNA and RNA to screen customers and orders to prevent the misuse of...
After fighting publicly, Sam Altman & Dario Amodei join hands; send letter to US Congress
Sam Altman and Dario Amodei have been engaged in a public conflict for years now. From disagreement over AI technology at OpenAI to Anthropic CEO openly criticizing the ChatGPT-maker’s contract with the US Department of Defense, the friction has become one of the most closely watched rivalries in the AI industry. In a latest, the two have come together along with other tech CEOs and leaders urging members of Congress to adopt new laws that would make it harder for bad actors to develop...
Whole-genome duplication shaped cell-type evolution in the vertebrate brain
Abstract The complex brains of vertebrates have more cell types than those of their closest relatives. Whole-genome duplications (WGDs) occurred during early vertebrate evolution1, but it is unclear whether the duplicated genes (ohnologues) facilitated cell-type evolution. Here using brain single-cell transcriptomes from five chordates—human2, mouse3, lizard4, lamprey5 and amphioxus—we report that many cell-type families with conserved core transcription factors in vertebrates do not show...
Gene ancestries reveal diverse microbial associations during eukaryogenesis
Abstract The origin of eukaryotes remains a central enigma in biology1. Continuing debates agree on the pivotal role of a symbiosis between an alphaproteobacterium and an Asgard archaeon2,3. However, the nature, timing and contributions of other potential bacterial partners4,5,6 and the role of interactions with viruses7,8,9 remain contentious.
Molecular glue degraders of HuR suppress BRAF-mutant colorectal cancer
Abstract BRAF gain-of-function mutations, particularly BRAF(V600E), affect roughly 10% of all patients with colorectal cancer (CRC), and portend poor prognosis with limited therapeutic interventions. BRAF inhibitors such as encorafenib are ineffective due to MAPK pathway reactivation driven by BRAF dimerization. Combined inhibition of BRAF and EGFR, although approved therapies, results in short survival benefits and frequent treatment resistance and relapse1,2,3.